fake websites selling cropped and docked dobermans for $800.00?
Sadly in today's digital world it's easy to copy photos from foreign and domestic Doberman breeders websites and then post them on your website's header as your own pretend Dobermans.
We have had our content copied and pasted onto some of these unscrupulous sellers websites and they were even too lazy to remove "Unique Dobermans" from the text!
We've had folks call and tell us our puppy photos were being used to sell puppies on dog selling websites such as puppy find or next day pets etc.
We have even had people steal our dogs photos and paste them onto their personal Doberman websites as their own dogs!
I'm sure you know you can't raise a puppy, remove the dew claws, dock it's tail and then crop it's ears all for $800.00.
Do your homework before you send money to someone with a pretty website.
Because if you think your getting this cropped and docked for $800.00...
How the Doberman Head Evolved..
Progress in Head Type and Size in Doberman Pinscher between 1899-1970THE FIRST THIRTY-FIVE YEARS. The first Standard ever written was by Germans in 1899, which read as follows: “General Appearance: The Doberman Pinscher should be built muscular and powerful, but not clumsy and massy, neither should he be greyhound-like. His appearance must indicate swiftness, power and endurance. Temperament should be lively and ardent.---Height at the shoulder: Males 21.6 to 25.6 inches; Females, 18.9 to 21.6 inches.—Length from occiput to first joint of tail about 27.5 inches.—Tail: cropped, not longer that 5.9 inches.”
“Head: Top of the head must be flat or may be slightly arched, but the forehead must be broad; stretched long, the head must go over into a not too pointy muzzle. Cheeks must be flat but very muscular. A dog of about 19.7 inches, height at the shoulder should measure about 16 inches around the forehead. The length of the head, from occiput to the tip of the nose should be 9.9 to 10 inches. Bite must be very powerful, well developed and closing right. Lips, lying close to the jaw, not drooping. The eye must be dark brown, medium sized, with an intelligent, gentle but energetic expression.---Ears cropped, not too short not too pointy.”
The above gives us a mental picture of the Doberman as presented by the German Standards of 1899, 1920 and 1925 which were in use up until the first American Standard in 1935.
In 1906 the Germans inaugurated comparative head measurements and the Doberman which then a short headed breed began its progress toward a long headed breed. The skull index in 1906 was .58 and was reduced in twenty five years to .42. It was during this period that the Manchester Terrier cross was made which contributed to better head type. The original infusions of Rottweiler and sporting dogs blood had contributed to a heavy head. The Doberman which influenced head type in this era were Leporello vd Nidda; Strumfried v Ilm-Athn: Feodor v Aprath; Leuthold v Hornegg; Waldo vd Strengbach; Sybille v Langen; Modern v Ilm-Athn; Lux vd Blankenburg: Favorit vd Konigstad and Lotte v Roeneckenstein. The goal of head type has been that of Favorit vd Konigstad which had a plainly marked stop; full muzzle, long, wedge-shaped head, correctly set eyes, thin, dry lips.
The first thirty five years after the adoption of the Standard saw really only two major changes. The dog increased about three inches in height—became a high legged animal; and by the lengthening of the head about an inch, became a long headed breed. At this early time statements were made that a domed head and triangular frontal bone were not desired.
THE SECOND THIRTY-FIVE YEARS. The first American Standard of 1935 added much detail and refinement of statement, also a list of faults. Relative statements were “The appearance is that of a dog of good middle size, with a body that is square--. Height at shoulder, males 24 to 27 inches, bitches 23 to 25 inches. Head: Long and dry, resembling a blunt wedge. Top of head flat, slightly depressed to ridge of nose, with nose extending as nearly parallel as possible to the forehead. Cheeks flat. Jaws full and powerful. Well filled under the eyes. Lips lying close to jaw. Eyes: Dark and of medium size, almond-shaped, with vigorous energetic expression.—Teeth strongly developed and snow white.—Ears: well trimmed and carried erect.”
There were no disqualifications at this time, under major faults were Missing Teeth, Undershot or Overshot exceeding one-quarter of an inch (imagine!), Shyness. Visciousness was just a fault. As the head was lengthened it became necessary to be on the alert for these conditions. Notice that we now recognize a taller dog and ask that be square. Along with more leg and longer head naturally came a longer neck with a slight arch.
In 1948 the Standard was revised again. “Height at the withers now 26 to 28 inches in dogs, ideal being 27; 24 to 26 inches in bitches, ideal being 25 ½. – Fearless, loyal and obedient” instead of loyal, obedient, fearless and aggressive. Asks for a head long and dry, resembling a blunt wedge with the words “both frontal and profile view” added. More detail on the head and then “scissors bite” and “22 teeth in the lower jaw and 20 in the upper”. The neck now specified as “upright, well muscled, well arched and length in proportion to body”. The word “upright” seems to have been somewhat misleading, as it brings the vision of “deer neck” or “ewe neck”, which is the result of poor front quarter angulation. For the first time now there are disqualifications: “Shyness, viciousness. Overshot more than 3/16 of an inch. Undershot more than 1/8 of an inch”. This Standard included a scale of points. Basic changes have been an acknowledgement of increase in size, stress of smooth lines and elegance and continued caution as to correct bites.
This era was the heyday of large kennels such as White Gate, Westphalia, Marienland and Ponchartrain which all did considerable importing of stock from Germany and Holland. From the combination of these various lines came a number of great dogs still familiar to us today, such as Uranus, Muck, Kurt vd Reinperle-Reingold, Jesse vd Sonnenhoehe and the later descendants such as Alcor, Dictator and Delegate—all of which have made their mark on today’s stock.
After a passage of twenty years, in 1969 the Standard was again revised and is in effect today. This Standard dropped the scale of points and the listing of faults. Statements remain approximately the same in regard to head, eyes, ears, but under teeth it now asks for “42 correctly placed teeth, 22 in the lower jaw, 20 in the upper” with a disqualification for four or more missing teeth. Viciousnes and shyness are no longer disqualifications, but dogs displaying these qualities are to be dismissed from the ring by the judge. Added details are given in the description of the body and gait and a 45 degree layback of shoulder is asked for.
From the above it becomes evident that fanciers feel that the progress within the last twenty years has been successful in regard to the overall balance, the station, the refinement and elegance of the Doberman, transforming him form a rather heavy appearing dog to one of great style and artistry: that some retrogression has taken place in teeth and mouths; that it is advisable to discourage further over-all size; that emphasis should now be on conformation for utility. The pendulum has swung a little too far in one direction, as usual, and in the search for beauty some factors necessary for working ability have been given minimum consideration.
One authority says that the shape of the head of a Doberman determines the dog’s type. Heads ARE important. The head is the dog’s weapon for use after arriving at the point of contact. The complete structure of the skull, muzzle and teeth enter into the picture for without the proper skull and jaw bones for the fastening of muscles, the catching, holding, lifting and crushing power is inadequate. As we ask for a longer head we must allow for a more prominent occipital bone for muscle anchorage. The depth and width of the attachment of the jaw bone and of the jaw bone itself must be in proportion to the over-all size of the dog. The plates forming the upper jaw bone and the fill under the eyes must be of sufficient width and length to be strong. Heads without sufficient depth from top of skull to bottom of jaw are weak; those which are too deep usually fall off in abrupt angles to pointy jaws. These have small teeth, narrow jaws, which are unserviceable.
Expression in an animal depends upon the shape of the head, the set and expression of the eyes, the carriage of the ears and this expression not only is unique to the breed but enables the knowledgeable beholder to know his character and disposition. Calmness, intelligence, gentleness and alertness show, as well as timidity, uncertainty and sharpness. If the head is too narrow the eyes are set too close together; if too wide, they are too round and face forward. The proper width and length of head with proper fill under the eyes gives the characteristic Doberman expression.
The Standard asks for a dog of medium size with a rear angulation to match a 90 degree front angulation. He must be compact, quick, agile -- characteristics which are not synonymous with great size. Take a look at the Standards of the really large breeds such as Mastiffs, Saints, Komondor,. Watch in the ring such breeds as Great Danes and Pyrenees and some of the diffiuculties of the extremely large breeds become evident. The Doberman Standard today is rightly trying to maintain the present size – to discourage any further advance in height.
We now have a dog “elegant in appearance, of proud carriage, reflecting great nobility and temperament.” This came about through breeders understanding that “Heredity is the transmission to the progeny of a trait which parents in their turn acquired from their ancestral genotype’, and in the application of the truth. They learned, among other things, that inbreeding produced slender, wiry, and larger body structure, thinner skins and finer hair. This progress was implemented by changing style which led to evolutionary improvements as “Inner hereditary traits and tendencies are called forth by external (environmental) influences and become fixed.” The present day Doberman is a dog of gentle curves, one into another, dry head and smooth dry body. At no point is he exaggerated. The good Doberman attracts attention over-all and in the discerning, grow in attraction. If any one point catches and holds the eye, it is probably overdone.
Gruenig says two things which would be well for all to remember; “No Doberman can possess “Adel” if its physical structure departs from utilitarian.”; and “Endow your litters with the priceless gifts of beauty and utility by a scientific selection of your breeding mate.”
If our best specimens today seem to be almost ideal, then it is up to the breeders to hold the ground gained and look forward to the wonders of the next thirty-five years.
Philpp Gruenig (1947)
Wm.Sidney Schmidt (1935)\
Wm. Sidney Schmidt (1940)
The Complete Dog Book (AKC) (1972)
Ruth McCourt (1974)
Children should always enjoy a higher social status in the family than the Doberman, but they may be too small and too young to be taken seriously as the dominant (or Top Dog) by the Doberman puppy or adult. Even a child who's taller than the family Doberman is not as well armed - or he or she had better not be.
The Doberman has much bigger teeth! Your child is relatively defenseless against the Doberman's weight, height and their teeth.
In a wolf pack, the young pups are outside the hierarchy. They certainly do not dominate any of the adults, but the adults defer to them and put up with all sorts of misbehavior because they are puppies.
An Alpha male wolf will carry food to his puppies and encourage them to take it from his mouth.
An Alpha female allows her puppies to climb all over her, to bite her tail and ears, to growl and snarl and act like puppies.
The Alphas protect the puppies from the youngest of their older siblings, the yearlings. Thus the subordinates learn to be similarly tolerant of the pups. This tolerance ceases abruptly when the pups hit adolescence, at around 6 months. From then on, it is up to each pup to find its place in the hierarchy, starting at the bottom. This is not a happy time in the life of a young wolf and most of them die, of starvation if not from dominant- inflicted wounds. So how do you apply this to your own families?
(Clue: Death is not an option.).
Do not expect a young child to dominate a Doberman or any dog, even a dog younger than the child.
Don't give the kid responsibility for your Doberman's behavior. Your child can learn to train the family Doberman, but an Alpha human must be present at all times, especially when both the dog and the child are young.
Food bowl train both the children and the dog, this is very important.
Children must leave the dog alone at mealtime unless Mom or Dad is right there. Do not tease a Doberman by attempting to control his food once it's been given to them.
If the dog is absolutely, positively steady on its Stay, the child can put down the food dish. Then step away when Mom or Dad says, before the dog is released. This tells the dog that the child has high status, but don't expect the Doberman to accept this. Stand by to assert your Alpha status and protect your child.
With adolescent humans, it's a judgment call (what isn't?) Some teenagers are really good with dogs and learn easily to train them. Others lose interest and wonder why the dog doesn't "listen" to them. The same teenager may be responsible sometimes and on another planet at other times. That's normal, human adolescent behavior. It is also normal for some adults, as well. Much training fails due to the OWNER's lack of patience. So nobody knows what to expect, including the dog. As a result the pooch learns to ignore or distrust the teenager. None of these outcomes is the fault of the dog. The Doberman is ultimately the responsibility of the adult humans of the family.
If you have a puppy, watch the relationships with the kids when the dog hits adolescence.
If your child has a good, buddy-type relationship with the Doberman, it's reasonable to hope it'll continue, but keep a parental eye on the situation.
If the dog starts pushing the limits (see Alpha Boot Camp Article), step in like an Alpha wolf with a yearling. You set the rules, and your adolescent dog is not allowed to come in as the Heavy with your kids. Period. NEVER, NEVER, NEVER allow a Doberman or any other dog and a young child (newborn through preschooler) to be together without an responsible person's hand within 6 inches.
Babies and puppies routinely hurt each other badly, for neither is old enough to know how not to.
The outcome of these episodes is not pleasant for anyone. The youngster learns to fear dogs, and the puppy learns to hate kids.
Keep your adult hands between your dog of any age and your baby.
Teach your baby to pat-pat the dog nicely and gently with an open hand, on its side or back.
Don't allow the child to touch the Doberman on the head or tail. Ears and tails are too easy to grab and pull; eyes can be poked with inquiring fingers. Keep the child's hands away from the underside of a male dog, for there are things to grab there, too. Praise your kid for doing it right. "You're a good dog petter!" may be more effective than "No, no!"
Teach your Doberman to lick the kids' hands but never to use teeth or claws. Be careful about faces. Dogs probably should not lick babies' faces, though they often want to very much for they are loaded with yummies after a meal. Be guided by your child's reaction. If the baby does not like having her/his face licked, don't let the dog do it, but if the kid loves it, OK.
Watch for teeth, if the yummies are dried on a bit. Pulling off hard, sticky yummies often requires teeth, from the Dobermans point of view. Mom or dad's hands must be right there.
Don't assume that anybody is going to remember these lessons for more than
a few seconds. BE THERE to protect your child and your Doberman puppy.
10 Ways to Stress Out Your Doberman
1. Punish him for behaving like a dog. Your Doberman is a creature of opportunity, and when you give him opportunities to "misbehave" by leaving tantalizing items within his reach, he'll take advantage. Translation: don't leave food or scraps where he can smell or see them.
Don't leave him in the bedroom alone with your slippers or socks. Insure the only opportunities you provide your Doberman are ones he can succeed at.
2. Tell her "no" over and over. If your Doberman is doing something she shouldn't be doing, telling her "no" will probably cause her to stop the behavior temporarily. But saying no to a Doberman without offering an alternative turns your "no" into merely an interruption, not a request or demand.
Unless you show your Doberman what you want her to do instead of what she's doing, chances are she'll be right back doing it sooner or later.
3. Give her a variety of verbal commands for a single behavior. Many pet guardians assume their Doberman knows English, and therefore knows that "drop it" and "leave it" mean the same thing, or that "get it" and "bring it to me" are the same.
Train your Doberman to respond to simple, preferably single-word commands (sit, stay, come, down, pee, poop, etc.) and then use those words, and only those words, to communicate. Otherwise, you'll create stress in your Doberman because she knows you want her to do something, but she doesn't know what based on the words you're using.
4. Tell him "it's okay." Many Doberman guardians tend to say this phrase to their pet when something decidedly not okay is about to happen. For example, you're driving your dog to the groomer, which is a place he's not fond of. You've driven him there often enough that as always, he starts to whine when the grooming shop comes into view.
And as always, you say, "it's okay" in an effort to comfort him. The second you utter those words he knows without a doubt where he's going, and his stress level shoots through the roof.
"It's okay" becomes a verbal cue to panic. Instead, help your dog learn to relax and cope with anxiety-producing situations.
5. Pull his leash. A dog that has been properly trained to walk on a leash doesn't typically do a lot of pulling, so if you feel the need to constantly yank the leash to redirect him, it's probably time to refresh your pet's leash manners.
It's also important to anticipate that your Doberman will naturally stop and sniff as often as possible, and pull in the direction of someone or something interesting. Be kind and understanding – allow your furry friend a reasonable amount of time to smell-inspect his outdoor territory and pick up his pee-mail without yanking him toward your destination.
6.Hold him while you hug or kiss him. Canines really don't get these human expressions of affection and can be confused by them – especially when the hugger or kisser is a relative stranger. Also, since Dobermans are typically being held (restrained) during the bear hug or smooch-fest, it increases their stress level.
Imagine how you would feel if someone large and in charge grabbed hold of you and wouldn't let go. It's not a good feeling and does nothing to generate trust, right?
Unless your Doberman is remaining contentedly still on his own while being hugged or kissed, it's best to stick to stroking and petting, which most dogs can't get enough of.
7. Stare at her. Most people are uncomfortable being stared at by other people, so it's easy to imagine how unsettling it might be for your Doberman. The canine species views staring as a confrontational sort of a "Let's get ready to rumble" signal, which naturally triggers a stress response. There's no need to stare at your Doberman unless you're returning her gaze.
8. Point or shake your finger at her. The finger pointing/shaking thing is a universal stress inducer for dogs (and many humans). That's probably because it's usually done while you're standing over your pooch in a menacing posture, or while you're speaking in a tone of voice that signals your displeasure.
Many a guilty dog look is the result of the finger-pointing thing, but your pet isn't so much feeling guilty as uncomfortable, wary, confused, and yes… stressed.
9. Tell him to "get down" when he jumps up. If like most people you use the verbal cue "down" to ask your Doberman to go from a sit to a lie-down, it's not going to work in situations where he's jumping up on you or someone else (or a piece of furniture). Train him to stop jumping with the verbal command "off" or "paws on floor" instead. You'll save your Doberman the confusion and stress that comes from trying to understand your command, and you'll potentially save yourself or a guest from a friendly mauling.
10. Wake her up. Unless there's a pressing reason to awaken your four-legged family member from a nice snooze, try to avoid it. Being shaken or shouted awake is stressful for all of us.
By Dr. Karen Shaw Becker
The warm summer months are upon us, and I know many of you have lots of outdoor activities planned that include four-legged family members. So this is a good time for a reminder to avoid foxtails when you’re out and about with your dog.
If you’ve never heard of them, foxtails are treacherous little plant awns that grow from the ear or flower of many types of grasses. They have hairy-looking little appendages that have spikes and sharp edges designed to attach securely to whatever or whoever happens by so they can spread their seeds to surrounding areas.
Foxtails grow all over California, have been reported in almost every state west of the Mississippi and are spreading to the east coast of the U.S. as well. There are several varieties, both native and non-native, but only some have harmful spurs. One dangerous variety is foxtail barley, which is found across the U.S. except in the south Atlantic and Gulf Coast states, and also grows throughout Canada and in parts of Mexico. Other harmful varieties include the giant foxtail, cheatgrass and ripgut brome.
In recent years, Midwestern states have seen a sharp increase in foxtail-related infections in dogs, especially sporting and hunting dogswho run through thick brush where they can inhale or swallow foxtails. Bottom line: if you live where foxtails grow, you and your dog can encounter them in parks, open fields, on city sidewalks and even in your own backyard.
Why Foxtails Are so DangerousIn late spring and early summer, foxtail plant heads turn brown and dry, and scatter across the landscape. The tiny spikes on the plant heads allow them to burrow into soil, and wildlife also helps spread them around.
Virtually any exposure your dog has to grass awns is potentially hazardous. The foxtails inevitably make their way into the noses, eyes, ears, mouths and just about every other opening of dogs' bodies, including the vulva and penis. They can get deep into your dog's nostril or ear canal or under the skin in no time, and often too fast for you to even notice them.
These deadly little plant heads can burrow into your dog's fur and pierce the skin, often between the toes. They can end up virtually anywhere in your pet's body, and symptoms depend on where the foxtail is located. For example, if your dog is shaking her head, there could be a foxtail in an ear canal. If she's suddenly sneezing uncontrollably, she could have one in her nose. Foxtails in the lungs can cause coughing and difficulty breathing.
A dog's body isn't capable of processing foxtails and can neither degrade nor decompose them. To make matters worse, foxtails carry bacteria and can only move in one direction (forward). Unless they’re found early, they can continue to travel throughout a dog's body, creating abscesses, damaging tissue and causing grass awn disease.
A grass awn infection can be very difficult to diagnose, in part because the infection occurs behind the migrating foxtail. In addition, foxtails are hard to see using traditional imaging techniques, because they are small, covered with infection and scar tissue, and are invisible on x-rays.
As you can probably imagine, once a foxtail is roaming around inside your dog's body, it can be incredibly difficult to find. It's not uncommon for veterinarians to perform multiple surgeries before a foxtail is finally located and removed.
Signs of a Foxtail Invasion and When It’s Safe to Remove ThemSigns that your dog may have encountered foxtails is sneezing if the invader got into his nose, pawing at the nose, and nasal drainage or infection. Foxtails that imbed in the skin typically cause inflammation, redness, irritation and oozing sores.
Other signs can include draining tracts (openings in the skin from which discharge drains), squinting, head shaking, excessive licking (especially the paws), scratching, chewing, lethargy, depression and loss of appetite. If a foxtail travels to the abdominal cavity, there can be fever and abdominal pain. Foxtails lodged in the vagina or urethra can cause pain and difficulty urinating.
If you see foxtails in your dog’s coat or anywhere on the outside of her body, including between her toes, remove them immediately either by hand or with a brush.
However, if you suspect or know there’s a plant awn in your dog’s nose or another body opening, or if you see an oozing sore or drainage tract, it’s best to take your dog to a veterinarian for removal. As I explained earlier, foxtails and other types of plant awns have spikes or hooks that dig into whatever surface they attach to, including flesh and tissue. Plant awns that are embedded in tissue are very tricky to remove because they can break apart, leaving a portion of the awn behind.
Not only does the remaining piece of the foxtail continue to cause inflammation and infection at the entry site, but it typically moves forward and deeper into the skin. It can potentially migrate throughout the body, ending up almost anywhere, including the lungs, abdominal organs, spinal cord and even the brain.
Protecting Your Pet From Foxtails and Grass Awn DiseaseOne of the biggest challenges in keeping your dog safe from foxtails can be learning how to identify them. They are usually a golden-brown color, but depending on the variety they can be green, white, yellow or dark brown, and can vary in size from about a half-inch to 3 inches in length, and one-eighth to a half inch in diameter.
To familiarize yourself with foxtails and other potentially dangerous plants where you live or visit with your dog, you can search the database at Center for Invasive Species and Ecosystem Health to see images of plants that grow in your area.
Obviously, avoiding foxtail exposure altogether should be the goal, but that's not always possible. If your dog does encounter foxtails, it's important to carefully comb through his coat, and also check his ears, mouth and between his toes a few times each day to remove any that you find before they have an opportunity to wreak havoc on your pet's health.
If your dog has a long coat and spends a lot of time outdoors, consider trimming (not shaving) his coat during the warmer months, and don’t forget the hair between his toes and pads. You might also want to check into these safety devices other dog owners have created to keep their canine companions free of foxtails:
Doberman Genetic Diseases and why you should know about them
Doberman health is a controversial topic in the doberman community worldwide. Despite modern diagnostic tools (DNA and medical tests) the longevity of the breed continues to decline due to Doberman genetic diseases. There are more and more Dobermans dying at a younger age.
Ability to sell puppies worldwide through social media had dramatically changed mentality of many breeders.
It seems that more breeders produce puppies purely for profit to supply the demand of online shoppers.
And why not? Be honest with yourself: when you see a cute puppy photo online – you are already sold.
And majority of puppy buyers won’t even look beyond that. So, why not sell more of those cute puppies if the demand is already high?
Some doberman genetic diseases are more common in certain types than others (read working vs show dobermans about different types of dobermans). And there are common diseases that are prevailing in the Doberman population worldwide. Breeders from different countries focus on different sets of Doberman genetic diseases. For American Doberman Pinscher the list is most likely to start with wobbler’s syndrome and von Willebrand’s disease. Western European Doberman (working line) breeders focus on thyroid. Eastern European Doberman breeders tend to disclose a variety of eye diseases.
Majority of breeders avoid mentioning the most fatal doberman genetic diseases. Perhaps, they don’t want to scare away potential puppy buyers.
Below is a comprehensive list of known Doberman genetic diseases in ascending order (from the least severe to the most fatal).
1. Genetic eye diseases. Eye diseases are more common in Eastern European Dobermans than American Doberman Pinschers (read american vs european doberman type). Dogs with genetic eye disorders are easily identified during ocular exam by an ophthalmologist.
PHTVL/PHPV (Persistent hyperplastic tunica vasculosa lentis/persistent hyperplastic primary vitreous) are rare disorders. The blood vessels on the back of the eye are not fully developed or deformed in fetus. This can cause small pigmentation (dots) on the back of the lens. In more severe cases – bleeding in the eye, cataract, blindness.
PRA (Progressive Retinal Atrophy) is an inherited disease of the retina, in which the rod cells in the retina are programmed to die.
Cataract – the lens of the eye becomes cloudy and eventually all functional vision is lost.
Hypoplasia/Micropapilla – optic nerve disease.
In years, that I’m observing the European Doberman breed, I came across only a few cases of serious eye issues in European Dobermans due to inherited eye diseases. Eye diseases are not fatal, but expensive to care for.
2. vWD (von Willebrand’s disease). It’s an inherited bleeding disorder caused by lack of certain protein (von Willebrand factor protein) that is responsible for blood clotting. The vWD disease is easily identified by the DNA test, performed once in a lifetime. The disease appears only if a dog has two copies of the gene. It’s a well known disease and easily controlled with selective breeding.
What you need to know about von Willebrand’s disease is that the entire Doberman population is divided into three categories:
– vWD-clear – don’t have the gene, don’t have the disease,
– vWD-carrier – have only one copy of the gene, don’t have the disease, and
– vWD-affected – have two copies of the gene and are affected by bleeding disorder.
Only vWD-affected dogs have bleeding disorder. Nearly half of the modern population of European Dobermans are vWD-carriers (have the gene, but not the disease). It is common to breed vWD-clear to vWD-carrier. Breeding carrier to carrier will produce partially affected litter (some puppies will be affected). vWD-affected dogs should not be bred to other affected or carriers.
In general, Dobermans develop only a mild form of this disease. So, even affected dogs can live long life with proper management of the disease. vWD-affected Dobermans are more common in Western European (working) lines, and American Doberman Pinschers. I have not come across a vWD-affected Eastern European dobermans.
If you wonder why not eliminate even vWD-carriers – the answer is: genetic diversity. Each gene is responsible for many traits than just one known disease. Eliminating vWD-carriers will wipe out a large genetic pool, that the breed so desperately needs. And there are breeders that are willing to breed vWD-affected dogs in order to diversify the population.
3. HD – Hip dysplasia (and elbow dysplasia – ED). In other words it’s a badly formed hip that restricts movements. The disease progresses as the dog ages. Hip and elbow dysplasia are identified with X-Rays when a dog is about 2 years old (when the bones are fully formed).
Usually, the hips are rated as Excellent (HD-A), good (HD-B), fair (HD-C), and downhill from there. Ideally, you want a dog with HD-A or HD-B rating.
Hip dysplasia was a big problem in the breed about 20-30 years ago. To the point that it was made mandatory for all dogs to send x-ray results to the main registry in Germany. Dogs with hip dysplasia were banned from breeding. This is still mandatory for German Shepherds since it’s an acute issue for that breed.
Hip issues still occur in Dobermans. So, when buying a puppy, just make sure that both parents were old enough when X-Rays were taken. Demand to see veterinary certificates. Dogs with hip dysplasia should not be bred.
4. Thyroid (Hypothyroidism). It’s a thyroid hormone insufficiency. Dysfunction of thyroid gland when it’s not producing enough hormone to maintain dog’s metabolism. Diagnosed by blood test analysis.
I didn’t come across thyroid issues in Eastern European Doberman, but it is more common in Western European Doberman (again referring to working vs show dobermans) and American Doberman Pinscher (read american vs european doberman type). For breeding purposes in Eastern European dobermans the test is performed once in a lifetime, but it is advised to repeat the test as the dog gets older. In Western European lines and American Doberman Pinscher – thyroid is tested more often (annually?).
Hypothyroidism is easily (and inexpensively) controlled with daily medications. And just like with von Willebrand’s disease – there are some breeders that are willing to breed dogs with thyroid insufficiency in order to diversify the gene pool. If left untreated – hypothyroidism can lead to serious autoimmune issues.
5. DM (degenerative myelopathy), CVI (cervical vertebral instability – aka Wobbler’s syndrome). DM and CVI are genetic spinal diseases. Degenerative myelopathy in dobermans usually affects rear limbs. Wobbler’s syndrome is specific to cervical vertebrae – a spinal cord compression that can lead to neck pain and paralysis. There is a DNA test for DM, performed once in a lifetime. I think CVI (Wobbler’s) is identified only after a dog shows signs of the disease.
It’s not a very common disease in Eastern European lines, but it happens occasionally. I more often see DM/Wobbler’s discussions about American Doberman Pinschers.
6. Cancer. Cancer in Dobermans is rapidly spreading, taking the second place as a cause of death in Dobermans worldwide. It is a silent killer that is difficult to spot until it is too late. There are no genetic tests allowing to screen Dobermans for genetic cancer predisposition.
As you probably know – some forms of cancer are environmental (acquired). Like lung cancer in people who smoke, or skin cancer in people who exposed to sun. In dogs, environmental cancer largely points out to commercially produced dog food (kibble). For that reason, there are many veterinarians advocating for home-made or raw diets for all dogs (not only Dobermans). Read about better dog food options.
Inherited cancer or cancer predisposition is clearly traceable from generation to generation in some bloodlines and can be tied to certain popular dogs in pedigrees. Unfortunately, cancer is not the problem number one for the Doberman breed and breeders often just ignore it when selecting mating partners.
7. DCM (dilated cardiomyopathy). This is a complex genetic disorder and is the leading cause of death of the Doberman breed worldwide. Doberman DCM is a genetic disease of a heart muscle which becomes progressively weak and eventually leads to heart failure. Dogs literally drop dead. DCM is the main reason for decreased longevity in the breed.
It is suspected that about 70% of Dobermans worldwide are affected by this disease (as of 2017). At the rate the disease is spreading, by 2040 , 100% of the Doberman breed will be affected. In some dogs the disease occurs at a later age (around 9 years old or older), but there are more and more dogs that die from DCM at a younger age. Many popular studs (that already produced hundreds of offsprings) get diagnosed at the age of 4 yo.
In a nutshell – the disease can only be reliably identified with annual heart tests – 24 Holter monitor and Echo-Doppler test. DNA test is inconclusive and as of 2018 still means nothing and are not reliable for breeding. Actively bred dogs must be tested with Holter and Echo-Doppler annually and within a year prior to mating.
So, if you see that parents of the litter are “Cardio free”, “heart tested”, “DCM DNA clear” – this means nothing. DEMAND to see authentic certificates (signed by a veterinarian cardiologist) for 24 Holter monitor AND Echo-Doppler (both, not just one). If the test is older than a year, if the breeder refuses to show you the certificates, if the breeder relies on the DNA test only – DO NOT BUY FROM SUCH BREEDER. You are more likely to end up with a puppy that will drop dead in a couple of years. Also, my advice – don’t buy puppies from very young parents. Genetic diseases tend to manifest themselves in mature dogs.
DCM is an acute issue for the breed and a complex topic that I’d like to discuss in a separate article in more details (the article is coming soon).
There are several other genetic diseases that appear in the breed, but they are rare and in most cases are the result of certain gene combination from both parents. Unfortunately, there are no screening for genetic compatibility in dogs to eliminate mating of dogs that can produce offsprings with these diseases:
– Autoimmune disorder is when dog’s immune system attacks its own cells. It is suspected that vaccination is the main trigger of autoimmune disorder, but it is unclear if some dogs are genetically predisposed to it.
– Kidney dysplasia is when kidneys don’t mature and at some point can no longer extract toxins from the body.
– Entropion – is an inverted eye lid.
– Epilepsy is a neurological disorder and causes abnormal brain cell activity.
Obviously, dogs that developed these diseases should not be bred. And parents, who produced such an offspring should not be bred together again (but can be bred to other dogs).
– Color dilute alopecia and albinism are specific to American Doberman Pinschers only. This is due to unregulated breeding practices in the US that allowed breeders to create variety of colors in Dobermans. It is in high demand among uneducated puppy buyers, who don’t know what health issues are behind multi-colored Doberman Pinschers. There are only two acceptable colors of Doberman in Europe – black and brown. In the US – there are also blue and fawn. Color dilute is genetic hair loss and skin issue that causes dog to suffer from severely dry skin, hair loss and bold patches. Color dilute is an essential disorder of every blue and fawn doberman. Albinism (white doberman) was also produced in the US and turned out to be an incredibly sickly type. Luckily, they now ban albino dobermans (and those, carrying the gene) from breeding.
Breeders will always breed. For decades they were unable to resolve minor (like eye disorders) or life-threatening health issues (DCM being the major one) in the breed. Unregulated breeding practices led to creation of more severe doberman genetic diseases. Doberman organizations worldwide are ignoring health situation in the breed, focusing on beauty contests and easy money. Doberman clubs in some countries don’t even regulate breeding practices, which allow breeders to put any two dogs together and profit from the result. While we – buyers and breed fanciers – have to watch our dogs suffer and die, spending thousands of dollars on vet bills. And until we – buyers and breed fanciers – are willing to blindly pay for a cute puppy – this will not change.
When looking for a puppy – make sure both parents are tested for all known genetic diseases. As you can see – doberman genetic diseases list is long. Don’t trust what’s written on the websites, social media posts or words of the breeder. Demand to see proper and all test certificates. If a breeder doesn’t show all or some of the health certificates – this should be alarming. If you leave health concern to chance – chances are high you will have to pay for it later.
Thank you: http://dobermanblog.com/doberman-genetic-diseases/
What is Cocci?
COCCIDIA are small protozoans (one-celled organisms) that multiply in the intestinal tracts of dogs and cats. They are most common in puppies and kittens less than six months of age or adult animals that are stressed or have a suppressed immune system.
Young Doberman puppies during birth are naturally exposed to their mother's feces, urine and any fecal load shedding from the stress of birth thru her milk.
If the mother is shedding the infective microorganisms in her feces from the stress of a new litter, then the young Doberman puppies will likely ingest the invisible protozoa and coccidia will develop within their intestines.
Most young puppies who are clinically ill from coccidia are two weeks of age and older. As a young animal ages, he tends to develop a natural immunity to the effects of coccidia and they are never thought of again....l
The primary sign of a puppy suffering from coccidiosis is diarrhea. The diarrhea may be mild to severe depending on the level of infection. Blood and mucous may also be present.
Severely affected animals may also vomit, lose their appetite, become dehydrated, or worse.
Fortunately, coccidiosis is treatable with A prescription drug called Ponazuril, if your veterinarian is still prescribing the dangerous and ineffective drugs such as Albon® and trimethoprim/sulfa please ask him to consider the newer and more effective drug Ponazuril
Because these drugs do not kill the organisms, but rather inhibit their reproduction capabilities, elimination of coccidia from the intestine takes time.
By stopping the ability of the protozoa to reproduce, time is allowed for the puppies own immunity to develop and remove the organisms.
To give your puppies digestive tract a boost while being treated, try Acidophilus+or Bene-Bac Pet Gel.
Most importantly start your puppy out on NuVet Vitamin Supplements
Rest assured your new Doberman puppy has NOT been raised in a stressful "Kennel" situation, however your Unique Doberman puppy has been raised naturally with the freedom to explore his outdoor world where he has been exposed to the native bacteria and protozoa that lives here in western America, some of which are of course Giardia and Coccidiosis.
Your puppy is free to explore and chase the Hawks, Eagles, rock chucks, cottontail bunnies, blue bellied lizards, white tailed deer, fox and coyote that may travel across our acreage.
This is why we strongly recommend that you continue your puppy on the great line of NuVet products that they and their parents have been on since before conception.
Use Order Code: 69685
To Order NuVet Toll-Free call: 1-800-474-7044
Use Order Code: 69685
International customers (including U.S territories) please call 800-474-7044 to order
Another reason your family should own a Unique Doberman..
"Earlier studies have shown that growing up on a farm reduces a child's risk of asthma to about half. We wanted to see if this relationship also was true also for children growing up with dogs in their homes. Our results confirmed the farming effect, and we also saw that children who grew up with dogs had about 15 percent less asthma than children without dogs. Because we had access to such a large and detailed data set, we could account for confounding factors such as asthma in parents, area of residence and socioeconomic status" says Tove Fall, Assistant Professor in Epidemiology at the Department of Medical Sciences and the Science for Life Laboratory, Uppsala University, who coordinated the study together with researchers from the Karolinska Institutet in Stockholm, Sweden.
"These kind of epidemiological studies look for associations in large populations but do not provide answers on whether and how animals could protect children from developing asthma. We know that children with established allergy to cats or dogs should avoid them, but our results also indicate that children who grow up with dogs have reduced risks of asthma later in life. Thanks to the population-based design, our results are generalizable to the Swedish population, and probably also to other European populations with similar culture regarding pet ownership and farming" says Catarina Almqvist Malmros, senior author on the study, Paediatrician at Astrid Lindgren Children's Hospital and Professor in Clinical epidemiology at Dept of Medical Epidemiology and Biostatistics at Karolinska Institutet, Stockholm.
A total of more than one million children were included in the researchers' study linking together nine different national data sources, including two dog ownership registers not previously used for medical research. The results are being published in JAMA Pediatrics. The goal was to determine whether children exposed to animals early in life are at different risk of asthma.
Materials provided by Uppsala Universitet. Note: Content may be edited for style and length.
Urinary Tract Treatment and Prevention:
Cranberry Extract is high in Vitamin C and prevents bacteria from adhering to the bladder wall.
1) Cranberry Extract—prevents adhesion of bacteria to mucosa, high in vitamin C, cooling properties
3000 mg of Cranberrier = 24 oz of juice
1/8-1/4 capsule TID cats & small dogs
½ capsule TID large dogs
1 capsule TID giant dogs
NO SIDE EFFECTS
2) Couchgrass (Crab Grass)—contains mannitol & mucilages, good for inflammation, mild antibacterial effect
3) Garlic—antibiotic, good for E. coli, Klebsiella, Proteus, Staph
Average clove of garlic has 100,000 Units of Penicillin
VERY HIGH doses can cause Heinz body anemia in cats
2-4 drops S-BID cats & small dogs
4-10 drops S-BID medium dogs
Garlitrin 1 tablet SID large dogs
4) Goldenseal—antimicrobial, stimulates immune system be enhancing macrophage activity
Caution is indicated in animals with low blood pressure, low blood sugar and pregnancy
Do NOT use for prolonged periods of time.
5) Horsetail—strong diuretic, soothing, anti-inflammatory, lithotriptic (dissolves stones)
Good for dogs with IVD and urine retention
6) Marshmallow Root—soothing and healing to inflamed mm., enhances WBCS, diuretic
7) Uva Ursi—antiseptic, diuretic, strengthens and tones
Do not use with concurrent GI problems or pregnancy,
long term use lowers K (potassium)
8) Vitamin C—antioxidant, enhances immunity, increases interferon, increases thymic hormones
During stress, the body secretes more vitamin C and makes more susceptible to infection
DOSAGE FOR ACUTE SYMPTOMS —Give every 2 hours to bowel tolerance for 24-48 hours
250-500mg BID small
500-750 BID medium
750-1000 BID large
1000-2000 BID giant
Water-soluble-->excess is excreted
ANIMALS’ APAWTHECARY—Standard dosing …
12 drops per 20 pounds—Glycerin-Based Formulations
Cigarette smokes contains cadmium--> promotes urinary stone formation
1) Vitamin B6—lowers production & excretion of oxalates:
2-10 mg/day small, 10-20 mg/day medium, 30 mg/day large, 40 mg/day giant
2) Magnesium—low levels increase calcium oxalate stone formation (phenonomena seen w/Struvite diets)
Lose Mg in stress situations, sulfate form can cause loose stool
50 mg SID small, 100 mg SID, medium, 300 mg SID large, 400 mg SID giant
3) Glucosamine—inhibitors of growth & aggregation of Ca+ oxalate crystals,
250-1000mg BID, NO side effects
4) Corn Silk—anti-inflammatory, diuretic properties, removes gravel from urinary tract
Infusion for large (1-2 oz) and giant (3-4 oz) dogs QID initially then BID
NO known side effects, prolonged use can irritate bladder
5) Gravel Root—powerful solvent, can be used alone or in combination, NO known side effects
Decoction-->3 ml TID small, 5-10 ml TID medium, 1-2 oz TID large, 3-4 oz giant
6) Wild Carrot—highly valuable for gravel, stricture or any obstruction in urinary tract, “ace in the hole”
No side effects, can be given to rabbits too
Infusion-->3-5 ml B-TID small, 5-10 ml medium, 15-20 ml large, 1 oz giant
What to Feed Your Unique Doberman for optimal health
Is your young adult or adult Doberman hyperactive, does he chase his tail bite at imaginary friends or run crazily thru the house and yards acting like a RACE HORSE?
Well, let me ask you... Are you Feeding your Puppy or Adult Unique Doberman like a Race Horse!
We here at Unique Dobermans feed a grain free high quality kibble along with steamed Normandy vegetables, cottage cheese, scrambled eggs, raw and cooked meat, pressure cooked poultry bones and all and any other high quality and unprocessed food. Our kibble is from Costco, their puppy food in the blue bag. We have also sucessfully fed our puppies Taste of the Wild.
Keep your Unique Doberman puppy in a healthy weight, if you don't know what that is please take him to your veterinarian, or better yet keep in close contact with your breeder (ME), you don't want an overfed puppy as it could lead to bone growth issues, nor do you want your puppy's growth stunted by being feed too little.
A Unique Doberman puppy will grow very rapidly, keep organic coconut oil on hand and give him a tablespoon over his food each day, this will help his coat and help keep his heart healthy as well as provide extra energy for those fast growing days.
Our Unique Dobermans are "Ranch" dogs first and foremost!
They run and hunt each day on our acreage where they may find a variety of dinner menu ideas running around.
Raw meat is natural and healthy for our Dobermans. We feed lots of raw beef with the bone on, this is great for our dogs coats and teeth. Big hardy bones give our dogs plenty of exercise and is a natural stress reliever.
We also "Free Feed" our active Dobermans. Each bedroom has fresh water in an auto water which comes directly from our 280 foot deep well, and each bedroom also holds a 35 lb bag of high quality kibble either from Natures Domain (grain free) or Taste of the Wild. (not ever more than 28% protein)
Our Dobermans are also feed steamed vegetables with pressure cooked poultry each week.
Each Unique Doberman is an individual with different needs and each is feed accordingly.
Our treasured mothers are fed a high quality diet which resembles our puppy diet.
Help! My Puppy Has Papilloma Viruse Warts
The most common methods of Natural Wart Removal include:
Home remedies for warts
EPA Announces Voluntary Cancellation of Toxic Chemical in Flea Collars
The U.S. Environmental Protection Agency (EPA) announced Friday that it has reached agreement with two major pet product companies to cancel flea and tick pet collars containing the insecticide propoxur. The agreement, with a long phase-out period, was reached between the agency and the two companies as a result of EPA’s risk assessment in fall 2013, which found unacceptable risks to children from exposure to pet collars containing propoxur.
The agency found that children were exposed to propoxur pet collars on the first day following application. Flea and tick collars work by leaving a pesticide residue on dogs’ and cats’ fur, which can be transferred to people by hugging, petting, or coming into contact with the pets. The major source of exposure to these chemicals is from absorption through the skin after directly touching the treated pet. Small children may ingest pesticide residues when they touch a treated cat or dog and subsequently put their hands in their mouth.
Under the cancellation agreement, Sergeant’s Pet Care Products, Inc. and Wellmark International will have until April 1, 2015 to continue producing the pet products containing propoxur under the trade names Bansect, Sentry, Zodiac and Biospot, and can continue to distribute them until April 1, 2016. EPA states that it will continue to watch for incidents from the use of these collars and is prepared to take further action if necessary.
Though this is a remarkable step towards removing a harmful product from the market, the extended phase-out period continues to allow children to be exposed. In fact, EPA has an astounding history of negotiated multi-year phase-outs with industry. As seen in other EPA decisions, cancellation of a toxic pesticide does not mean that the chemical would be removed from the market, but it is allowed to linger on the market for years continuing to threaten human health and contaminate the environment.
Propoxur is a carbamate insecticide first registered in the U.S. in 1963 for the control of household pests. Despite the fact that it was banned in 2007 for indoor uses to which children would be exposed, it remained widely used in flea and tick collars. EPA completed the propoxur pet collar risk assessment in fall 2013 in response to a 2009 Natural Resources Defense Council (NRDC) petition to cancel the uses.
A 2011 study published in the journal NeuroToxicology found a positive link between exposure to the pesticide propoxur and poor motor development in infants. At the age of two, children exposed to propoxur in the womb experience poor development of motor skills, according to a test of mental development. Propoxur can be very dangerous to humans and the environment. Common symptoms of poisoning include malaise, muscle weakness, dizziness, and sweating. Headache, nausea, and diarrhea may also result. EPA considers propoxur a possible human carcinogen, while the state of California classifies it as a known human carcinogen. Propoxur is also highly toxic to beneficial insects such as honey bees as well as crustaceans, fish, and aquatic insects.
Source: EPA Press Release
#1 Weed and Feed Products Threaten Human Health, and are Especially Dangerous For Children
A growing body of scientific evidence continues to confirm the widespread health
effects of Weed and Feed products. 2,4-D, the pesticide in most Weed and Feed products,
is a neurotoxicant and contains half the ingredients in Agent Orange. Studies show that
exposure to 2,4-D is associated with neurological disorders, reproductive problems,
kidney/liver damage, non-Hodgkin’s lymphoma and other cancers, and disruption of
the endocrine (hormonal) system.1 Children are especially at risk for increased
exposure to Weed and Feed since they play on lawns for extended periods of time and
put their hands and other objects into their mouths.2 In general, children are most
susceptible than adults to pesticides because they take in more chemicals relative to
their body weight than adults and they have developing organ systems that are more
vulnerable and less able to detoxify chemicals.3 Endocrine disruptors are of particular
concern for children because, depending on timing, minute doses can effect the
function of cells and tissues and cause problems during critical growth stages.
Disruption of the endocrine system is associated with a range of developmental
problems including deficient brain function, learning disabilities, and other problems.
Exposure to herbicides such as 2,4-D is not limited to the outdoors. Studies have
shown that lawn chemicals drift and are tracked indoors where they may remain in
carpets and on surfaces for up to a year when not exposed to direct sunlight.4 A single
turf application of 2,4-D can remain inside the home at exposure levels ten times
higher than pre-application exposures.5 In a 2003 study of indoor air toxins, 2,4-D was
detected in the dust of 63% of sampled houses.6
#2 Weed and Feed Hurts Dogs and Wildlife
2,4-D has been shown to have negative impacts on a number of animals. Studies have
found that dogs whose owners use 2,4-D lawn products are twice as likely to develop
canine malignant lymphoma.7 The latest EPA assessment of 2,4-D acknowledges the
susceptibility of dogs to poisoning by 2,4-D and other lawn pesticides but does not
propose any label warnings to users.8 Wildlife is also negatively affected by Weed and
Feed. Exposure to 2,4-D has shown to reduce hatching success and cause birth defects
in birds.9 Studies also show 2,4-D products to be toxic to earthworms that are vital to
healthy soil, and to have negative impacts on beneficial insects, such as honeybees,
predatory beetles, and ladybugs. 10,11
#3 Weed and Feed Pollutes Drinking Water Sources
Since Weed and Feed combines a fertilizer and an herbicide, it directs the user to spread
the herbicide throughout the lawn instead of just where weeds are present. Most users
are believed to overuse Weed and Feed products, not realizing that it actually contains a
pesticide or just by thinking that more is better. This is exacerbated by the fact that
only around half of households actually read and follow the label carefully when
using pesticides and fertilizers.12 Since 2,4-D is highly mobile in soil13 the overuse of
Weed and Feed products leads to runoff that contaminates groundwater and
watersheds. Studies by the U.S. Geological Survey show 2,4-D is the number one
herbicide most frequently detected in streams and shallow ground water throughout
the country from home and garden use.14 2,4-D has also been detected in groundwater
in at least five states and Canada.15 Partially due to the problem of overuse and water
contamination, the Canadian Medical Association passed a resolution calling for the
ban of Weed and Feed products.16
#4 Local Governments Are Calling For a Ban on Weed and Feed
As part of EPA’s assessment of 2,4-D for reregistration in March 2005, the public was
able to submit comments. Over 1000 letters calling for the cancellation of Weed and Feed
products were received by the agency including some from local governments and
state and local agencies such as Seattle Public Utilities, the California Regional Water
Quality Board, Clark County(Washington), and King County(Washington).17
#5 We Don't Need Weed and Feed
Weed and Feed is not an effective solution to weed maintenance. It can actually damage
the health of lawns by harming microorganisms, beneficial insects, and earthworms
that are essential to maintaining healthy soil and therefore, healthy turf. Typically,
weeds cover a small fraction of lawn area, and any herbicide applied to weed-free
areas is wasted. Even if a lawn contains as much as 50% weeds, then half of the
herbicide is unnecessary and contributes to runoff and health risk without providing
any benefit. There is no need to expose the public to this toxic chemical in the water,
the air and the soil when safe and effective alternatives exist. Examples of alternatives
to 2,4-D include corn gluten as a safe pre-emergent general herbicide, vinegar to
selectively kill certain weeds, weeder machines that simply use hot water or heat, long
handled mechanical weed pullers, and pulling out weeds by hand. Natural organic
fertilizers or slow-release fertilizers help to maintain a healthy lawn.
Doberman – Melanoma, Lipoma, Histiocytoma, Fibroma, Myxoma, Primary brain tumor
Your pet’s chances of acquiring both bladder cancer and lymphoma dramatically increase if your pet is exposed to certain lawn and garden products. The lawn and garden care chemical most notorious for being toxic is called 2, 4-D, and is almost surely in your weed killer product among others. Aside from 2, 4-D, you’ll want to avoid products with Carbary, Pronamide, Chlorothalonil, or Maneb, common pesticide components which seem to correlate closely with increased cancer risk.
According to the report “Reducing Environmental Cancer Risk: What We Can Do Now” by the President’s Cancer Panel established in 1971): “The entire U.S. population is exposed on a daily basis to numerous agricultural chemicals, some of which are also used in residential and commercial landscaping. Many of these chemicals have known or suspected carcinogenic or endocrine disrupting properties. The EPA has required testing of less than 1 percent of the chemicals in commerce.”
Pesticides and Autism:
Mount Sinai is leading an effort to understand the role of these toxins in a condition that now affects between 400,000 and 600,000 of the 4 million children born in the United States each year.
"A large number of the chemicals in widest use have not undergone even minimal assessment of potential toxicity and this is of great concern," says Dr. Landrigan. "Knowledge of environmental causes of neurodevelopmental disorders is critically important because they are potentially preventable."
CEHC developed the list of ten chemicals found in consumer products that are suspected to contribute to autism and learning disabilities to guide a research strategy to discover potentially preventable environmental causes. The top ten chemicals are:
4. Organophosphate pesticides
5. Organochlorine pesticides
6. Endocrine disruptors
7. Automotive exhaust
8. Polycyclic aromatic hydrocarbons
9. Brominated flame retardants
10. Perfluorinated compounds
In addition to the editorial, the other four papers also call for increased research to identify the possible environmental causes of autism in America's children. The first paper, written by a team at the University of Wisconsin -- Milwaukee, found preliminary evidence linking smoking during pregnancy to Asperger's disorder and other forms of high-functioning autism. Two papers, written by researchers at the University of California -- Davis, show that PCBs disrupt early brain development. The final paper, also by a team at UC -- Davis, suggests further exploring the link between pesticide exposure and autism.
A study presented in the January 2012 issue of the journal Environmental Research concluded that exposure to professionally applied lawn pesticides was associated with a significantly (70 percent) higher risk of canine malignant lymphoma (CML).
It’s a broad conclusion and light on specifics. The case-control study, conducted between January 2000 and December 2006 at the Foster Hospital for Small Animals at Tufts University’s Cummings School of Veterinary Medicine, was structured around a 10-page questionnaire that was mailed to dog owners who were having their pets treated at the Foster Hospital; the resulting data came from the owners of 266 dogs with confirmed cases of CML and 478 dogs in two control groups (non-CML cases).
The questionnaire was not included in the article; a summary stated that it covered a wide variety of data considerations, including breed, weight, medical history, and the types of chemicals used in the home. The characteristics of the CML cases did not vary much from the controls, other than in the weight category (the CML dogs tended to weigh more than 50 pounds). Exposure to types of flea and tick products and frequency of administration was similar among the groups, as was overall exposure to lawn care products.
What did show cause for concern was that the CML cases were more likely to live in homes that reported professionally applied pesticides and herbicides, though the results were only marginally significant for the herbicides. Exposure to other types of professionally applied lawn care products was not associated with increased risk. There was an increased risk, however, for dogs who live in homes where owners applied lawn-care products containing insect growth regulators – substances that inhibit the development of insect eggs and larvae.
One disappointment: specific lawn care chemicals or insect-growth regulators were not identified. Instead, the umbrella categories of herbicide, pesticide, insect growth regulators, fungicide, rodenticide, and fertilizer were used. It could be that some of these chemicals are already designated as known carcinogenics. The article notes that studies evaluating frequency of exposure and exposure dose are needed; thus it appears that the researchers did not determine which chemicals the dogs were exposed to, in what quantities, or for how long.
Also disappointing was the fact that genetic factors were apparently not considered as part of the study. Three-fourths of the CML dogs were classified as purebred, as was the control group. The incidence rate of CML is not the same for all breeds; increased risk has been reported for several breeds including Basset Hounds, Boxers, Airedales, Golden Retrievers, Saint Bernards, Bulldogs, Bullmastiffs, and Scottish Terriers. This predisposition could indicate an inherited characteristic.
Like the canary in the mineshaft, dogs can serve as sentinels for human disease because they are our close companions and are subjected to many of the same environmental influences. Canine cancers have the same biology and behavior as human cancers, and in some cases have identical histology and response rates to treatment. The goal of this study was to identify risk factors for CML from exposure to environmental chemicals in an effort to provide insight to risk factors for humans in developing non-Hodgkin’s lymphoma.
Keep in mind that exposure to lawn care products is different for canines than it is for humans. People can know if a lawn has been recently treated with chemicals and thus avoid it and take precautions when handling such chemicals. Our pets have no such option; their uncovered and unprotected bodies come in direct contact with the environment. They see an enticing outdoor carpet, perfect for rolling around on, running across, playing fetch and wrestling with playmates on, and even ingesting. Dogs have their mouths on everything: themselves (grooming), their playmates, their toys and sticks lying in the grass, and yes, the grass itself. And those mouths can be the conduit from external to internal exposure.
Though more study is needed, the preliminary findings of this study suggest that you can reduce your dog’s risk through the following:
-Don’t use pesticides on your own lawns, or allow lawn-service providers to use them on your property.
-Don’t use lawn care products that contain insect growth regulators.
-Prevent your dog from walking on (or rolling on, eating, etc.) any lawns, unless you are able to determine that absolutely no pesticides are used to maintain them. (Most municipalities are required to make their chemical lawn-care regimens available to the public. It says something about these chemicals that their use is prohibited on most public school grounds.)
– Barbara Dobbins
Minnesota becomes the first state to ban the toxic antibacterial pesticide triclosan in consumer personal care and cleaning products statewide. Read the press release, and learn more about triclosan here.
Dog owners need to realize that many of the common chemical herbicides are responsible for a large number of pet poisonings. Herbicides can have both short- and long-term effects. They can cause an array of problems that range from mild vomiting to cancers and death.
The Environmental Association for Great Lakes Education (EAGLE) sites the “Journal of the American Veterinary Medical Association” and the “Journal of the National Cancer Institute” in their conclusion that dogs exposed to lawns treated with herbicides can double their chances of developing canine lymphoma. Herbicide use can increase the risk of canine bladder cancer in some breeds by as much as seven times.
When purchasing herbicides, always read the labels so that you understand the dangers associated with the product. The labels include information about the individual chemicals used in the herbicide and safety information that can help protect you and your pet. The labels also specify the amount of time that you need to keep your pet away from plants that have been sprayed. Pay close attention to this information. It is not there for decoration. It can save your dog’s life.
There are several dangerous chemicals found in herbicides. One is arsenic, which causes vomiting, diarrhea, pains in the abdominal area, coma and death.
Paraquat is another herbicide that has proven to lead to death.
Metaldehyde, which is generally found in snail and slug baits, has been associated with abdominal cramps, vomiting, tremors, seizures and death. One of the big problems associated with this herbicide is the fact that metaldehyde tastes good to dogs.
Disulfoton, which is still used to protect roses, is part of a class known as organophosphates. While many organophosphates have been pulled from the market, disulfoton, which is also used in fertilizers that contain bone meal and blood, attracts dogs. Unfortunately, it is still used despite the fact that it can make pets extremely ill and can lead to death.
Roundup, a commonly used herbicide, also presents dangers to your pets. Dogs exposed to the polyethoxylated tallowamine (one of the inert ingredients) in it can experience extreme vomiting and should be seen immediately by a veterinarian. Follow the directions and do not let your dog walk in areas where the herbicide has been sprayed for at least a day.
Most chemical herbicides are the most dangerous when they are wet. The threat becomes less when the herbicide application dries completely. However, dogs that ingest weeds sprayed with herbicides can still become very ill and death can occur.
Don’t assume that your yard will be safe even if you don’t apply herbicides. Your yard can become contaminated with the chemicals from spray that blows in from a neighboring property. If your municipality has an herbicide spray program, your yard can easily become contaminated from an herbicide application.
If you are on good terms with your neighbors, discuss the dangers of herbicide use with them. If they still insist on using the dangerous chemicals, hopefully they will notify you before they apply them so that you can take the proper precautions. If you know spraying is likely to occur, it is important to keep your dog indoors. Do not let toys or food dishes remain outdoors to become contaminated with the herbicide products.
If your neighbor or municipality embarks on a spraying program, ask that they give you 48 hour’s notice before applying herbicides. This will give you ample time to make arrangements to keep your dog from being exposed.
If you rent a property and the landlord cares for the yard, you need to discuss an herbicide application schedule with the person in charge. Again, ask for advance notice so you can be sure your dog is not outdoors when the spraying is done.
If you suspect that your pet has come into contact with freshly sprayed herbicides, call a veterinarian or pet poison control center immediately. Be prepared to provide as much information as possible so that the vets know how to treat the dog. Different poisons require different antidotes.
Try organic herbicides whenever possible. Vinegar is fast becoming one of the most popular organic herbicides. According to the Sustainable Agricultural Systems Laboratory, which is part of the USDA, vinegar has an 80 to 100 percent kill rate in concentrations of 10 to 20 percent. The vinegar you purchase from the grocery store contains only five percent acetic acid and will not work well as an herbicide. Visit your local garden store or check the Internet for vinegar that has higher concentrations of acetic acid.
You might also try using corn gluten meal in place of chemicals. Corn gluten meal (CGM) is a natural substitute for synthetic pre-emergence herbicides. Pre-emergence herbicides attack seeds while they’re still in the ground, before the seedlings emerge from the soil. CGM is a by-product of commercial corn milling that contains the protein fraction of the corn. Its use poses no health risk to people or animals. In fact, because it is 60% protein, corn gluten meal is used as feed for cattle, poultry, fish, and dogs. In addition to the 60% protein, corn gluten meal is 10% nitrogen, by weight.
Corn gluten meal is a relatively new herbicide that was discovered during turf grass research at Iowa State University. The study showed that corn gluten meal prevented grass seeds from sprouting. The meal works to stop the germination of seeds. It does not kill or stunt existing plants.
Corn gluten meal is available in powder or pellet forms. The meal should be applied late in April or early in May and again in mid-August for best results. The corn gluten meal should be applied at a rate of 20 pounds per 1,000 square feet of lawn. Water lightly after application to activate the corn gluten meal.
As more Americans move toward a greener lifestyle, more information is being published on organic herbicides. Fortunately, more commercial sprayers are offering organic treatments of weedy lawns. Don’t be afraid to ask for natural alternatives.
8 Tips for Preventing your Kids and Dobermans to unseen exposure to Allergy and Cancer Causing Toxins and Poisons.
1. Your kids may be more profoundly affected by random, chronic pesticide exposures than adults simply because they are smaller and still growing, according to studies.
Kids also tend to have a more severe reaction to gases, toxins and pesticides. Study after study shows this can cause extreme harm to your growing child, their bodies absorb gases and toxins like a sponge, thereby contributing to various skin issues and rashes, lung issues such as asthma and in severe cases, dizziness, disorientation and even death.
In 2005, the Organic Consumers Association gathered evidence showing that cumulative exposure to residues on foods from pesticides — some of which, like 2,4-D, are the same as those used on lawns — could affect child development. The OCA summed up:
“According to the Environmental Protection Agency (EPA) and the National Academy of Sciences, standard chemicals are up to ten times more toxic to children than to adults, depending on body weight. This is due to the fact that children take in more toxic chemicals relative to body weight than adults and have developing organ systems that are more vulnerable and less able to detoxify toxic chemicals.”
Similarly, Beyond Pesticides notes:
“The National Academy of Sciences reports that children are more susceptible to chemicals than adults and estimates that 50 percent of lifetime pesticide exposure occurs during the first five years of life.
EPA concurs that children take in more pesticides relative to body weight than adults and have developing organ systems that are more vulnerable and less able to detoxify toxic chemicals.”
Findings showing that kids take on a proportionately heavier load of chemicals — from food, plastics, and household products — has led to concerns that lawn chemicals contribute to their “body burden,” in the parlance of those who discuss personal pollution issues.
Scientists are finding that pesticides, like certain components in plastic such as BPA, can disrupt the human hormone system. Both major weed and feed chemicals, atrazine and 2,4-D, are considered to be “potential endocrine disruptors” (along with an array of other pesticides), meaning they can interfere with the human body’s hormonal system.
Endocrine disruptors are known for their insidious way of creeping into the human body undetected at low levels, essentially thwarting the body’s usual defenses. They mimic or interfere with natural hormones, acting on the thyroid or pituitary glands, reproductive organs and the brain.
Studies suggest that this interference can cause problems with child or fetal development, and metabolism and fertility, later in life.
The effect of synthetic endocrine disruptors on reproductive organs could explain why human fertility issues are suddenly “off the Richter scale,” said Mark Kastel of the Cornucopia Institute in a recent interview.
“Sometimes exposures to these toxics can have catastrophic lifelong impact,” he said. “It might be a triggering device, especially in the reproductive organs, causing them to develop inadequately.”
Endocrine disruption is just one way pesticides may harm kids.
Two studies by Minnesota researchers found that pesticides, either a mix that included 2,4-D but also fungicides and glyphosate, appeared to raise the risk of birth defects among the children of exposed farm families.
Other studies have found that the use of household and lawn pesticides raises the risk of childhood leukemia (here’s one paper that reviewed 17 studies of the link to leukemia). Beyond Pesticides has collected a long list of research, showing implicating pesticides in childhood development issues, increased asthma rates and even brain cancer.
Cerera was a very large and fast growing puppy! She rapidly grew to the height of 29 inches tall. Luckily we escaped the common problems associated with a fast growing puppy. However you may not be so lucky!
Panosteitis is sometimes called "growing pains." Panosteitis may occur in more than one bone at a time and often moves around from leg to leg.
The limping usually occurs very suddenly and spontaneously, or without a history of trauma or excessive exercise.
Pano usually presents itself around 5 to 6 months of age and can come and go even moving from leg to leg up until your Doberman puppy is 18 months of age.
Some tips we can share with you if your Doberman puppy limping are:
1. Make sure your puppy receives a thorough lameness examination by a competent Veterinarian to rule out tendon or joint pain from an injury. Although panosteitis is not a serious disease, and is a common cause of lameness, other, more serious bone diseases can cause lameness in young dogs. In order to be sure that a sudden onset of lameness is not caused by one of these more serious bone diseases, radiographs must be taken. If the radiographs show the typical lesions of panosteitis, then you can rest assured that your dog will eventually outgrow the problem.
2. Although there are potential links between diets containing excessive levels of dietary protein and/or calcium, it isn't recommended to feed large breed puppies with an adult dog food that contains lower levels of protein and calcium. The reason for this is that many grocery store brands of adult dog food also have lower calories or energy levels than puppy food. This will actually cause your Doberman puppy to eat a lot more of a low-energy food to meet their growth requirements. Eating more of a low energy diet will result in a higher overall intake of protein and calcium which can disrupt their delicate growth ratio.
A better option is to feed your Doberman puppy a high quality diet that has been specifically formulated for use in large breed puppies or adolescents, and to restrict the quantity fed to keep the dog at a lean, healthy body weight. Do not allow your puppy to become overweight.
3. Episodes can last for two to three weeks or can continue for months at a time. The dog may show hesitance to walk, run, jump, or exercise. If the affected bone is squeezed, the dog will exhibit pain as well.
Some dogs run a low-grade fever during episodes of panosteitis. Others have elevated white blood cell counts. The condition typically affects the radius, ulna, humerus, femur, and tibia, but once in a while the condition can affect the foot and pelvic bones as well.
4. Large Dog Breeds at higher risk of Panosteitis:Large fast growing breeds have a higher risk of Panosteitis. Panosteitis is especially common in German shepherds. Other dog breeds where this condition is quite common are the Great Dane, the Doberman pinscher, the Labrador retriever and the Golden retriever.
Signs and Symptoms of Panosteitis in dogs:
The classic signs and symptoms of Panosteitis in Doberman puppies is lameness. Panosteitis can affect different bones at different times, and you might notice how your Doberman puppy seems sore and lame in one hind leg only to have it suddenly switch to the opposite leg seemingly overnight! Such a cycle of lameness can last from 2-3 weeks for each leg, and your Doberman puppy will often experience periods when it has no apparent symptoms at all and happily runs and plays like any other Doberman puppy.
If your Doberman puppy is suffering from Panosteitis it can develop a fever or experience nausea, become lethargic and even lose its appetite.
Panosteitis is not cured surgically or by prescribing any particular drug.
The treatment of choice for Panosteitis is simply rest and exercise restriction, a Doberman puppy with Panosteitis must be given plenty of rest, you may even need to crate them at times.
If your Doberman puppy is in a lot of pain, your vet can may prescribe a non-steroidal anti-inflammatory drug to help make them more comfortable. (NO Rimadyl)
Most Dobermans fully recover from Panosteitis without any permanent side effects.
MSM - directions are on the bottle for the size of the dog. Open the capsule and sprinkle the powder on the food.
Herbsmith Comfort Aches - available in a tablet and a powder.
Yucca Intensive - 10 drops in food. This is bitter, so mix it into the food (shake the bottle well).
Vitamin "E" 1 Capsule daily.
Vitamin "C" 500 mg daily.
I had been telling Bruce how we needed to get some weight off of one of our young females, we switched her to adult food but it wasn't quite soon enough and she has shown a bit of pain in her hind legs. Nothing serious but a wake up call to be on the lookout for Pano!
We have never had a serious case of Pano in our Dobermans and we are not wanting to experience any Doberman puppy with it now!
Remember to keep your young rapidly growing puppies lean and on a well balance diet. Do not feed extra calcium as this may disrupt the delicate balance between calcium and phosphorous a sure fire way to set your Doberman puppy up for a painful bout of Pano!
Our dogs are in the midst of an epidemic. It’s not an epidemic of viral disease, but of chronic ill health. They’re besieged with itchy, pus-laden, scabby skin; vomit and diarrhoea are the norm. One in every hundred dogs suffers from epilepsy, and an even higher number lives with painful arthritis. Allergies are also reaching epidemic proportions: dogs are becoming allergic to life.
According to Dr Jean W Dodds, an eminent vet and researcher, both allergic and autoimmune diseases have been rising since the introduction of modified live virus vaccines. Autoimmune diseases are where the body attacks self; they include cancer, leukaemia, thyroid disease, Addisons, Grave’s disease, autoimmune haemolytic anaemia, rheumatoid arthritis, diabetes, lupus, thrombocytopenia, organ failure, skin inflammations, and more.
We also seem to have a tremendous number of dogs with behavioural problems, largely due to over-vaccination and processed pet food. Vaccines are known to cause inflammation of the brain, as well as lesions throughout the brain and central nervous system. The medical term for this is ‘encephalitis’, and vaccine’s role is acknowledged in the Merck Manual. Merck is a vaccine manufacturer.
Years ago, I was the typical ‘responsible’ dog owner. My four Golden Retrievers were vaccinated every year, and they were fed a ‘complete and balanced’ pet food, recommended by my vet. The red carpet was metaphorically rolled out once a fortnight, each time I visited with a dog suffering from chronic disease. Eventually the problems became more serious: my dogs started to die years before their time.
Over the years, I’ve collected research documents to help me make decisions about my dogs’ husbandry, and to share what I’ve learnt with other dog lovers. I also hoped that vets would take notice of the research, and stop over-vaccinating. All medical interventions come with a risk – even the humble aspirin can be deadly. So you have to do a risk/benefit analysis whenever you consider medications. What, then, are the risks of vaccines?
Research by Frick and Brookes shows us that vaccines can trigger atopy (skin allergies). (Am J Vet Res. 1983 Mar;44(3):440-5). Dr Jean W Dodds tells us that retroviral and parvoviral diseases, and MLVvaccines, are associated with lymphoma, leukaemia, organ failure, thyroid disease, adrenal disease, pancreatic disease, and bone marrow failure.
Vaccines cause cancer in cats at their injection site and, according to the Journal of Veterinary Medicine, August 2003, vaccines cause cancer in dogs at their injection sites. Vaccines cause autoimmune haemolytic anaemia (JVM, Vol 10, No. 5, September/ October 1996; Merck Veterinary Manual), andarthritis (BVJ, May 1995 and Am Coll Vet Intern Med, 2000; 14:381). Epilepsy is a symptom of encephalitis, which, as we already know, can be caused by vaccines.
According to Dr Larry Glickman and his team at Purdue University, serum and foreign proteins in vaccines can cause autoimmunity (i.e. cancer, leukaemia, organ failure, etc.). This research also indicates that genetic damage is possible, since vaccinated dogs developed autoantibodies to attack their own DNA. Research from the University of Geneva echoes this finding.
Over the years, many vets, particularly in America, have been saying that they think vaccines cause a diverse range of problems in animals. For example, Christine Chambreau DVM said, ‘Routine vaccinations are probably the worst thing we do for our animals. They cause all types of illnesses but not directly to where we would relate them definitely to be caused by the vaccine.’ She is not alone in this view.
So imagine my dilemma ten years ago, when Edward and Daniel came into my home. Having already seen my vaccinated dogs suffer with chronic illnesses, and dying from cancer and leukaemia – knowing that vaccines may have caused these illnesses – what was I to do?
I concluded that I would rather risk viral disease with my dogs, than have them suffer from the epidemic of chronic and fatal illness that is gripping the canine population. I appreciate that some will consider me irresponsible. But what actually are we running from when we vaccinate?
OK, so distemper is so rare that most vets haven’t seen it in at least ten years. Also, according to the top researchers, and stated by the American Veterinary Medical Association, once immune to viral disease, dogs are immune for years or life. So why are vets and vaccine manufacturers still trying to get us to vaccinate against viral disease every year, or even three-yearly – especially when you consider the risk?
According to the Intervet data sheet, dogs will develop permanent immunity to hepatitis over the age of 12 weeks. So why keep vaccinating against that? Kennel cough is easily treated in most cases, and the vaccine isn’t very effective. So what’s the point? Leptospirosis is rare (my vet tells me he hasn’t seen it in ten years, either), and the vaccine is associated with some of the worst adverse reactions. Isn’t this vaccine an unacceptable risk, then? And parvovirus is – according to the Concise Oxford Veterinary Dictionary – rarely a problem for the normal healthy adult dog.
The next question, of course, is how do you get yourself a normal healthy adult dog? Aha – catch 22. In my view, you get a healthy adult dog by not vaccinating at all! Vaccines destabilise the immune system, leading to all sorts of chronic illness. From all I’ve seen and read, vaccines do not set your dog up for good health. They have the potential to make your dogs itchy, scratchy, vomiting, diarrhoea-filled, sickly, sub-normal shadows of their former selves – ready and waiting for the more serious killers like cancer to arrive. Vaccines represent the perfect recipe for the chronic illness epidemic I’ve been describing.
Does this mean I’ve left Edward and Daniel open and unprotected against viral disease? No. When they were puppies they were given the homoeopathic nosode, a safer vaccine alternative. They have also been fed naturally all their lives, providing vital nutrients to boost their immune systems, and they are exercised well (which also boosts the immune system). Have they ever they suffered from recurrent hot spots, allergies, digestive upsets, eye and ear infections, or any other chronic illnesses? No. Did they die of cancer at the age of five, or leukaemia at the age of six, or paralysis at the age of four – as my vaccinated dogs did? No. In fact, they’re probably very well equipped, and healthy enough, to withstand the diseases I might otherwise have vaccinated against.
Is probably good enough? Well – it’s the best anyone is going to get. Because even vaccines cannot guarantee immunity.
So am I taking the high risk option? I don’t think so. It seems to me that good health is a God-given natural right. It’s only man who messes it up. The natural order is wiser than any of us, and those of us who don’t vaccinate our dogs are proving natural law to be right.
by Catherine O'Driscoll in Vaccine Articles and News
The duration of immunity for Rabies vaccine, Canine distemper vaccine, Canine Parvovirus vaccine, Feline Panleukopenia vaccine, Feline Rhinotracheitis, feline Calicivirus, have all been demonstrated to be a minimum of 7 years by serology for rabies and challenge studies for all others.
In the Duration of Immunity to Canine Vaccines: What We Know and What We Don’t Know, Proceedings – Canine Infectious Diseases: From Clinics to Molecular Pathogenesis, Ithaca, NY, 1999, Dr. Ronald Schultz, a veterinary immunologist at the forefront of vaccine research and chair of the University of Wisconsin’s Department of Pathobiological Sciences, outlines the DOI for the following vaccines:
Minimum Duration of Immunity for Canine Vaccines:Distemper- 7 years by challenge/15 years by serology
Parvovirus – 7 years by challenge/ 7 years by serology
Adenovirus – 7 years by challenge/ 9 years by serology
Canine rabies – 3 years by challenge/ 7 years by serology
Dr. Schultz concludes: “Vaccines for diseases like distemper and canine parvovirus, once administered to adult animals, provide lifetime immunity.” “Are we vaccinating too much?” JAVMA, No. 4, August 15, 1995, pg. 421.
Yet vets continue to vaccinate annually. Dog owners feel that their vets are doing their dogs a great service by vaccinating every three years instead of annually – why do we allow it when these studies were done over thirty years ago and have been replicated time and again by other researchers?
Ian Tizard states: “With modified live virus vaccines like canine parvovirus, canine distemper and feline panleukopenia, calicivirus, and rhinotracheitis the virus in the vaccine must replicate to stimulate the immune system. In a patient that has been previously immunized, antibodies from the previous vaccine will block the replication of the new vaccinal virus. Antibody titers are not significantly boosted. Memory cell populations are not expanded. The immune status of the patient is not enhanced.
After the second rabies vaccination, re-administration of rabies vaccine does not enhance the immune status of the patient at one or two year intervals. We do not know the interval at which re-administration of vaccines will enhance the immunity of a significant percentage of the pet population, but it is certainly not at one or two year intervals. Tizard Ian, Yawei N, Use of serologic testing to assess immune status of companion animals, JAVMA, vol 213, No 1, July 1, 1998.
“The recommendation for annual re-vaccination is a practice that was officially started in 1978.” says Dr. Schultz. “This recommendation was made without any scientific validation of the need to booster immunity so frequently. In fact the presence of good humoral antibody levels blocks the anamnestic response to vaccine boosters just as maternal antibody blocks the response in some young animals.”
He adds: “The patient receives no benefit and may be placed at serious risk when an unnecessary vaccine is given. Few or no scientific studies have demonstrated a need for cats or dogs to be revaccinated. Annual vaccination for diseases caused by CDV, CPV2, FPLP and FeLV has not been shown to provide a level of immunity any different from the immunity in an animal vaccinated and immunized at an early age and challenged years later. We have found that annual revaccination with the vaccines that provide long-term immunity provides no demonstrable benefit.”
Why then, have vets not embraced the concept of lifelong immunity in dogs?
“Profits are what vaccine critics believe is at the root of the profession’s resistance to update its protocols. Without the lure of vaccines, clients might be less inclined to make yearly veterinary visits.Vaccines add up to 14 percent of the average practice’s income, AAHA reports, and veterinarians stand to lose big. I suspect some are ignoring my work,” says Schultz, who claims some distemper vaccines last as long as 15 years. “Tying vaccinations into the annual visit became prominent in the 1980s and a way of practicing in the 1990s. Now veterinarians don’t want to give it up.”
The report of the American Animal Hospital Association Canine Vaccine Taskforce in JAAHA (39 March/April 2003)3 includes the following information for vets:
Misunderstanding, misinformation and the conservative nature of our profession have largely slowed adoption of protocols advocating decreased frequency of vaccination'; ‘Immunological memory provides durations of immunity for core infectious diseases that far exceed the traditional recommendations for annual vaccination.
‘This is supported by a growing body of veterinary information as well-developed epidemiological vigilance in human medicine that indicates immunity induced by vaccination is extremely long lasting and, in most cases, lifelong.’
Both the AAHA and the AVMA must do more to “step up to the plate” says noted immunologist, Dr. Richard Ford. But the reality is the vets do not have to listen to the AAHA or the AVMA and it appears the state veterinary medical boards are not interested in enforcing vaccine schedules, opting to leave it up to the individual vet.
Dr. Bob Rogers hired a Chicago based law firm and initiated a class action suit for pet owners who were not given informed consent and full disclosure prior to vaccination administration. His article entitled “The Courage to Embrace the Truth”, states “While attending conferences like WSVMA and NAVMC I have asked over 400 DVMs from various parts of the country if they attended the seminars on New Vaccination Protocols. I was told by all but one, “I don’t care what the data says, I am not changing.” One DVM here on VIN even said “I am not changing until the AVMA makes me change.”
It seems that pet owners are against the wall when it comes to vaccination. The obvious conclusion is that pet owners who are concerned about the long term health of their companion animals must take it upon themselves to research vaccines, duration of immunity and vaccine dangers. At the very least, question every vaccine that goes into your animal – but none of the above information indicates you will get an honest or well-informed answer.
Be your dog’s advocate – protect him with knowledge and by taking a stand against unnecessary vaccination. His life may depend on it!
I came across this article I thought I'd share with you on how many times you can breed your female safely.
HOW OFTEN TO BREED YOUR BITCH
The experts all seem to agree that the least healthy approach for a breeding bitch is to skip heat cycles and keep the bitch unbred. This is not only Dr. Hutchinson's philosophy as Dr. Threlfall at Ohio State Univ. teaches the same thing (my husband just attended a Cont. Ed seminar on Canine Repro earlier this year at OSU). This is NOT new information, either. I was reading Dr. Billinghurst's book GROW YOUR PUP WITH BONES, which addresses the health of puppies as well as their parents and reproductive issues. This is not a new book (maybe 10 years old?). He states the same thing. Canines are meant to be pregnant on every heat cycle.
As Dr. Hutchinson explains it in his seminars, the hormones are the same and the bitch goes through the same changes whether they are bred or not. So when the hormones 'do their thing' to a uterus that does not have pups, it is "hammered" (his term) by the hormones and causes aging and thickening which makes the uterine lining less conducive to implantation and more prone to infection over time. The recommendation it to breed them young, breed on every heat cycle until you are done, then spay them. THAT is the healthiest scenario for your breeding bitch. While Dr. Threlfall and Dr. Hutchinson don't see eye to eye on some issues, this one they completely agree on. I have to wonder if anyone has found a vet knowledgable on repro issues who states otherwise.
Yet there are still people who refuse to believe this advice. I have often wondered about the practice of condemning back-to-back breedings. I wonder if it stems from the way bitches blow their coat post weaning which may lead people to feel the bitch is not recovering well. I know that our girls blow their coat at the same time they would after being in heat (about 4 months) whether bred or not, but the post puppy coat loss is usually more. I suspect that this appearance made people believe that the bitch was completely run down and it "was hard on her" having the pups.
Unfortunately, in our current PC environment, we want to suggest that people who breed more than one litter every several years are simply money hungry puppy mills and some of us are quick to condemn their practices based on this mentality. So if someone follows the EXPERTS advice concerning their dogs, the self appointed Ethics Police talk poorly of them ignoring the fact that what they are doing is biologically in the BEST interest of their dogs.
I think many people want to act like dogs are little people in fur coats. They want to suggest that what we may feel is how a dog feels. While I wouldn’t personally want to have a new child every year, I do believe that my dogs have always adored having puppies. Granted, there are certainly reasons why some bitches should probably not be bred again. Some are poor mothers. Some don't produce much milk. Some can't whelp or conceive w/o veterinary intervention. But the bottom line is that in a healthy normal bitch, breeding every heat cycle for as many litters as you want from that bitch, then spaying her, is the most healthy way to go. And that is from the people who are qualified to say so.
You know, cattle are kept pregnant every year starting when they would "freshen" (have their calf) at 2 years of age. They breed them until they won't breed anymore. If a cow is "open" (not pregnant), the farmer either tries to get her bred or sells her because wintering an open cow is a big money loser. Yes, it is certainly a business having calves (no one denies that), but the cattle certainly seem fine being pregnant all but three months of the year and well into their teen years. Just as an aside, cows/heifers start having calves at 2 years of age (earlier and they aren't fully grown so often can't calve on their own). They are bred back EVERY year. I know cattle is a money business and many of the Doggy PC Police want to say that breeding more than a few litters a year is only out of greed, but cattle NEVER get a break and apparently have no ill effects as a result. Also, dairy cows won't have milk unless they are bred back each year. But my point is that this does not seem to effect their health in a bad way at all and has been the way cattle have been kept for many many decades. If you tried to tell them that it is too hard on the cow to be pregnant every year, they would think you were a COMPLETE idiot!
The bottom line is that if you are a breeder… well, you breed! Perhaps it is time for some of us to rethink our beliefs that dogs should get a break between heat cycles for their health because under normal circumstances, this is simply not true.
As always, I encourage anyone with ideas on issues I’ve discussed, or issues they would like to see addressed, to please share their thoughts with me. I can be reached at email@example.com Thanks so much!
Doberman Puppy Bottle Feeding Recipe
There are times when either your new mother had a very large litter, is ill, has had an emergency c-section or passed during or shortly after birthing that you will need to supplement your puppies by tube feeding or bottle feeding.
We use whole goat's milk (not pasteurized cow's milk) from a local goat dairy, Whole goat's milk is by far the best to use. You can purchase goats milk from a local goat dairy (check craigslist.com in your area) Health Food Stores (they told us about the dairy), Fred Meyer and Walmart both sell evaporated milk, whole goat's milk and evaporated goats milk. Make sure you are using EITHER evaporated milk OR whole goat's milk. Do NOT use sweetened condensed milk or any type of cows milk!
Puppy Formula Recipe:
10 oz RAW Goats Milk
3 oz. sterilized water (baby water OR boiled water that is then cooled). This is NOT needed if using raw whole goat's milk.
1 raw egg yolk.
1 cup of plain yogurt (avoid skim or fat free if at all possible).
1 Teaspoon of liquid probiotics available at http://www.revivalanimal.com/
1/2 tsp Karo Syrup or Corn Syrup (NOT HONEY!)
1 Tablespoon of Coconut oil warmed into liquid form.
Blend 1/2 the milk with the egg yolk, yogurt, coconut oil and Karo syrup in a blender or use a wire whisk. After it's blended thoroughly add the rest of the milk and whiz once.
Place all the Doberman Pinscher puppy formula milk replacement into a clean container and refrigerate.
To use, warm puppy formula to body temperature around 101 degrees in a baby bottle placed in hot water, sprinkle a few drops on your inner arm to make sure it's not too hot! Discard any unused formula.
Make sure to poke a few extra holes in the newborn nipple so the thick mixture flows thru easily, but not so many or large enough holes that you choke your puppy.
Here at Unique Dobermans, we use cocounut oil as a coat conditioner, all around supplement for extra energy and for cardio health.
Trying to sort thru all of the supplements can be a confusing and difficult task for many Doberman owners, most dog owners have heard of the benefits of feeding fish oil but not coconut.
Coconut oil consists of more than 90% saturated fats, with traces of few unsaturated fatty acids, such as monounsaturated fatty acids and polyunsaturated fatty acids. Most of the saturated fats in coconut oil are Medium Chain Triglycerides (MCTs). The main component (more than 40%) of MCTs is lauric acid, followed by capric acid, caprylic acid, myristic acid and palmitic. Coconut oil also contains about 2% linoleic acid (polyunsaturated fatty acids) and about 6% oleic acid (monounsaturated fatty acids).
Most of the coconut oil benefits come from the MCTs. For example, the lauric acid in coconut oil has antibacterial, antiviral, and anti-fungal properties. Capric and caprylic acid have similar properties and are best known for their anti-fungal effects.
In addition, MCTs are efficiently metabolized to provide an immediate source of fuel and energy, enhancing athletic performance and aiding weight loss. In dogs, the MCTs in coconut oil balance the thyroid, helping overweight dogs lose weight and helping sedentary dogs feel energetic.
According to Dr. Bruce Fife, certified nutritionist and naturopathic doctor, coconut oil gently elevates the metabolism, provides a higher level of energy and vitality, protects you from illness, and speeds healing. As a bonus, coconut oil improves any dog’s skin and coat, improves digestion, and reduces allergic reactions.
Fed regularly to pets, coconut oil may have multiple benefits:
Nothing seemed to be helping Buffy – an affectionate buff colored Cocker Spaniel. His owner was interested in holistic avenues. She was referred to a dermatologist after she had tried everything, (even steroids) and nothing had helped. The dermatologist prescribed Atopica for Buffy and the improvement was almost immediate. She couldn’t understand why her holistic veterinarian and her local veterinarian (who was aware of her holistic leaning) had not prescribed Atopica.
The truth is that many veterinarians, both holistic and conventional, shiver with dread when they contemplate this product called Atopica. One of my clients – a nurse – contacted me after her veterinarian had suggested Atopica. She explained to me the strict rules and protective actions that a nurse needs to take when administering cyclopsporine, (which is the active ingredient in Atopica) to human patients were enough to make her very wary of using the product on her dog.
Atopica: What is it? Atopica is a product that touts its efficaciousness with dogs who have allergies and other dermatological conditions. Most of my readers will know that allergies result from an incorrect response of the immune system to foods and environmental substances. For many years steroids such as prednisolone were used to decrease the ability of the immune system to function and thus relieve the allergies. It’s when steroids can’t and don’t work that Atopica is prescribed.
Why does it often work so well? What does it do? How does it work?
Cyclosporine is a mycotoxin. Mycotoxins are harmful products produced by fungi. They are chemical in nature and are immune suppressing. Fungi rely on the mycotoxins they produce to kill any bacteria, other fungi, viruses and anything else that might compete with them. They suppress the immune system of dogs, cats and humans. Some examples of mycotoxins found in nature are aflotoxins, the most potent carcinogen on earth, and ocharatoxins – both produced by an Aspergillus fungi. Medical mycotoxins include Adriamycin, a chemotherapy drug and lovastatin, a cholesterol lowering drug.
The immunosuppressive effects of cyclosporine were discovered in Switzerland in 1972 and it was used successfully in preventing organ rejection in kidney transplants and later in liver transplants. Apart from transplant medicine, cyclosporine is used for a variety of skin conditions in both humans and pets. Of course, in transplant patients it suppresses the immune system so they do not reject their transplants.
The Side EffectsThe side effects of this drug include headaches, nausea, vomiting, diarrhea, shaking hands, swollen bleeding gums, cancer, kidney failure, hypertension, easy bruising, hearing problems, yellowing of skin and eyes, loss of consciousness, vision changes, swollen glands, immune suppression and dizziness. Interestingly, these aren’t side effects at all, but rather symptoms of poisoning caused by this mycotoxin poison. Farmers are familiar with the deleterious effects that mycotoxins can have on animals that eat moldy grain containing mycotoxins and the symptoms all agree. In fact, death is one side effect listed on the feline prescription.
The literature for people states that their risk of infection will be higher when they are on this drug and to avoid people with contagious diseases or infections. Of course your pets will have the same increased risk for infection and cancer. You’re told to wash your hands after you apply it to your cat or dog in the instructions. Hell, I’d wear latex gloves if I ever had to apply the product.
Here’s an interesting tidbit. I was totally amazed when I heard it and it made my distrust of the pharmaceutical industry go way up. I had a client who worked in the laboratories of a prestigious pharmaceutical company doing tests to determine the side effects from many drugs. She told me that they do initial testing protocols to set up the study and watch when the side effects occur.
Let’s say, for example, that after 90 days 40% of the rats develop cancer and 20% go into liver failure. This information results in the company designing the study to last for no more than 60 or 70 days so that they can state that after that time they found that a minor percentage had minor side effects.
I guess when you view it from their perspective it’s the smart way to do it.
It took many years of my practicing medicine to fully understand that drug companies want to make money and not help or cure patients. If one pill would cure your high blood pressure what would happen to their monthly revenue if you stopped going in every 4 weeks to refill. Just so, no pet vaccination company brags that their vaccinations last for 10 or more years because of the revenue that they would lose.
Nowadays, one out of two dogs will develop cancer. Every dog and cat needs an immune system that works and works well. And this is why veterinarians who care and also understand the mechanism behind how Atopica works shiver in their shoes. They wouldn’t use it on their dogs and cats and you shouldn’t use it on yours.
For many of my veterinary friends, just the mention of this product makes us stare at each other in disbelief- speechless. That’s why I decided to write this for you today. Then you’ll know what we know.by Dr. Deva Khalsa in Holistic Care
Diagnosis of DCM
Overt disease: When the disease is in the symptomatic or overt stage, diagnosis is not difficult. Symptoms may include shortness of breath, coughing, poor appetite and weight loss, fainting episodes. Physical examination will reveal a heart murmur, a gallop (an extra heart sound which indicates heart failure) and often irregular beats. Chest X-ray will show an enlarged heart and fluid in the lungs. Echocardiogram (ultrasound of the heart) will show a dilated left ventricle and atrium, with poor function and mitral valve insufficiency (leaking of the valve).
Occult disease: When the disease is in an asymptomatic or occult stage, the diagnosis may be suspected by testing, but at this date these criteria are not uniform from center to center. Echocardiography of healthy Dobermans recently studied at the UDC nationals and it was found that the average fractional shortening of these healthy dogs was 26% using a short axis view, and 22.5% using a long axis view.
In other breeds a fractional shortening of 25% or less in the short axis view is considered abnormal. This either indicates that a large percentage of healthy Dobermans have occult DCM or that the Doberman heart at baseline is not comparable to that of most breeds.
As such, to diagnose occult DCM, most centers now require not only a depressed fractional shortening, but some evidence of functional impairment such as dilated heart chambers or frequent ventricular arrhythmias.
While diagnostic criteria may vary from center to center, the largest ongoing study is being performed by Dr. Michael O'Grady at the University of Guelph, and his criteria are the best defined and studied to date.
To diagnose occult DCM, Dr. O'Grady requires the following echocardiographic findings, measured in the parasternal long axis view/ A fractional shortening of less than 20% with the following left ventricular chamber measurements: greater than 49 mm at the end of diastole (when the ventricle is completely filled), greater than 42 mm at the end of systole (when the ventricle has completed emptying).
This diagnosis is collaborated by a progression of 3-5 mm since a prior examination.
Alternatively, the diagnosis can be suspected by frequent ventricular arrhythmia – the criteria currently in use by Dr. O'Grady is greater than 50 PVCs (premature ventricular contractions) per hour (1200+ per 24 hr holter), however this study is on-going. This is measured by a Holter monitor, which is and ECG running continuously for a 24 hour period.
Duer et al. ( 4), who examined 50 young dogs of different breeds, reported the occurrence of II° atrio-ventricular block in one dog only. In another study performed on 44 Doberman Pinscher dogs, the only observed disturbance was a wandering sinus pacemaker in one dog (5). In 16/51 dogs (31.4%) no other arrhythmias were observed, similar to other authors’ findings ( 4- 6, 12, 17).
In conclusion, the mean 24-h heart rate frequency in healthy dogs is 100 beats per minute (range 80-120 beats/min), the mean maximum heart rate frequency is 210 beats per minute (range 180-240 beats/min), and the mean minimum heart rate frequency is 43 beats per minute (range 33-53 beats/min).
Healthy dogs may manifest single premature supraventricular beats up to 1,500/24 h, and single premature ventricular beats not exceeding 100/24 h. Furthermore, RR pauses not longer than 3 s may occur (Table 2). No impact of breed, age or sex on the analysed parameters was noted.
Weight Conversion Chart for Tube Feeding Puppies OR Bottle Feeding Puppies:
There are times when your newborn puppies mother is either unwilling or unable to properly nurse and care for her newborn babies, she may have had complications requiring a C-Section and if she was spayed her milking and mothering hormones have been removed with her ovaries. Maybe she has developed eclampsia and the puppies need removed, or maybe she has mastitis and they need removed or worst case scenario she died after whelping. Possibly it's something as simple as a very, very large litter of big thriving Doberman puppies, regardless of the reason here is a handy chart for determining how much formula your puppy should be consuming each day. Please divide the amount of formula into 6 equal feedings which will ensure the adequate amount of food being fed to your puppies every 4 hours.
If you don't own a weight scale now is the time to purchase a good digital scale, you will need to weigh your puppies every 12 hours to make sure they are gaining. Remember this is just a guideline, not a set in stone formula, adjust according to your puppies breed and rate of growth. And WEIGH THOSE BABIES!
CLICK HERE FOR GOATS MILK PUPPY FORMULA
1/2 ounce formula = 15cc
1 ounce formula = 30cc>
1 ounce = 28.3 grams
1 oz. = 8.3 grams 15cc daily
2 oz. = 56.6 grams 15cc daily
3 oz. = 84.9 grams 15cc daily
4 oz. = 113.2 grams 30cc daily
5 oz. = 141.5 grams 30cc daily
6 oz. = 169.8 grams 45cc daily
7 oz. = 198.1 grams 45cc daily
8 oz. = 26.4 grams 60cc daily
9 oz. = 54.7 grams 60cc daily
10 oz. = 83.0 grams 75cc daily
11 oz. = 311.3 grams 80cc daily
12 oz. = 339.6 grams 90cc daily
13 oz. = 367.9 grams 90cc daily
14 oz. = 396.2 grams 105cc daily
15 oz. = 424.5 grams 105cc daily
16 oz. = 452.8 grams 120cc daily
17 oz. =481.1 grams 120cc daily
18 oz. = 509.4 grams 135cc daily
19 oz. = 537.7 grams 135cc daily
20 oz. = 566.0 grams 150cc daily
21 oz. = 594.3 grams 150cc daily
22 oz. = 622.6 grams 165cc daily
23 oz. = 650.9 grams 165cc daily
24 oz. = 679.2 grams 180cc daily
25 oz. = 707.5 grams 180cc daily
26 oz. = 735.8 grams 195cc daily
27 oz. = 764.1 grams 195cc daily
28 oz. = 792.4 grams 210cc daily
29 oz. = 820.7 grams 210cc daily
30 oz. = 849.0 grams 225cc daily
31 oz. = 877.3 grams 225cc daily
32 oz. = 905.6 grams 240cc daily
33 oz. = 933.9 grams 240cc daily
34 oz. =962.2 grams 255cc daily
35 oz. = 990.5 grams 255cc daily
36 oz. = 1018.8 grams 270cc daily
37 oz. = 1047.1 grams 270cc daily
38 oz. = 1075.4 grams 285cc daily
39 oz. =1103.7 grams 285cc daily
40 oz. = 1132.0 grams 290cc daily